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Immunology, Ideology, Power

In 2005, experiments showed that injecting

the spike protein from a corona virus caused

lungs to fail (Kuba, et al.), and that antibodies

formed to the spike protein damaged the lungs

(Lin, et al.). For several decades, it has been

common knowledge that autoimmune disease

(such as “rheumatic fever”) could result from

the cross reaction of antibodies with microbial

antigens and antigens on the person’s tissues.

Recent studies are confirming the risk of

autoimmune disease from antibodies to the

covid spike protein. One cross-reaction with

the spike protein is the lung surfactant protein

(Vojdania and Kharraian, 2020; Talotta, 2021).

Cross-reactive antibodies and autoimmune

processes are also involved in cancer and

infertility.

With both the spike protein and the

antibodies to it producing such deadly effects,

using it in a vaccine to protect against the

corona virus would have seemed crazy to

anyone following immunological and virologi-

cal research after 2005, and the thought of

implanting a nucleic acid to cause the body to

produce the spike protein would have seemed

like criminal insanity at that time. Just 12 years

later, big corporations and their government

supporters found it convenient to forget the

recent science. For example, in 2017 the chief

medical officer of the Moderna corporation

gave a talk about what a nifty and simple idea

if would be to cause the body to produce its

own vaccine. Describing the process, he said

“So, here’s all the biology you need to know

in 30 seconds.” For the corporations and the

public health officials, that’s all the biology

they want to know, and all they want the

public to know.

In the immunity that results from interac-

tions with pathogenic organisms, and in the

immunity produced by a traditional vaccine

made from killed or weakened pathogens, the

body forms antibodies to many parts of the

potentially harmful organism. The result is

that we accumulate a great variety of antibod-

ies to multiple pathogens, and in the case of

corona viruses, children accumulate additional

immunity each time they have a cold. This

means that almost all children and adults are

immune to a great variety of corona viruses.

At birth, there are natural antibodies, that

developed along with the differentiation of

tissues. Naturally, they don’t inactivate the

body’s own tissues; they persist through life,

but decrease with aging, increasing the

susceptibility to infections and to loss of

normal functions (Palma, et al., 2018). They

apparently serve to protect tissues, recognizing

and eliminating deviations from normal

(Britschgi, et al., 2009). Their existence has

been generally accepted only in recent

Ray Peat's Newsletter

. . . it is easier to bamboozle than to debamboozle. Norman Angell

Copyright 2021 Raymond Peat P.O. Box 5764 Eugene OR 97405

September 2021

Not for republication without written permission.


Page 2

decades; their existence creates difficulties for

the dominant theory of immunity.

With natural antibodies, the issue of cross-

reactive antibodies and autoimmunity doesn’t

exist, but when the specialized adaptive

antibodies are produced, cross-reactivity is

always a problem—other organisms vary in

their tissue antigens, and antibodies binding to

those will have varying degrees of cross-

reactive affinity for our own tissues. For

example, the lactobacilli that thrive in women

during their fertile years have some antigens

that resemble human connective tissues, and

it’s possible that they are a factor in diseases

such as lupus and rheumatoid arthritis that are

prevalent in women especially during those

years. Any infection or vaccination, especially

when there’s an excess of estrogen or a

deficiency of antiinflammatory factors, can

permanently damage your health.

The first two people in the U.S. reported to

have died from “covid,” at the end of

February, 2020 were in their 80s and 90s, in an

extended care home in the Tacoma, Washing-

ton area, and within a few days a third death

was also said to have been caused by covid;

their deaths were attributed to covid on the

basis of a PCR test that, despite not having

been validated as tests normally are, had just

been authorized for use under a newly adopted

Emergency Use Authorization. Secretary of

Health and Human Services, Alex Azar, had

declared a Public Health Emergency on

January 31, 2020. The fact that the WHO

recommended, and the CDC ordered, the

operation of the test at 40 to 45 cycles, known

to produce mostly false positive results was

widely noticed: as testing increased nationally

beginning in March, the incidence of “cases of

covid” increased at the same rate as the testing.

The “test epidemic” served to create an eager-

ness to get the inoculation. Exactly a year after

the WHO had begun promoting the testing

method of Christian Drosten, with instructions

to misuse it, the inoculation campaign was

underway, and the WHO changed its instruc-

tions, reducing the number of cycles—with

the obvious intention of creating an impres-

sion that the inoculations were lowering the

rate of infection, as the number of false

positive results was immediately reduced.

In the late 19th century Paul Ehrlich,

working with the German dye industry,

noticed that different bacteria and tissues

varied in their affinities for coal tar dyes. He

reasoned that the ability of some dyes to kill

certain pathogenic organisms was analogous

to specific immunity, such as demonstrated by

vaccination. He extended his dye-binding

theory to the use of arsphenamine (Salvarsan,

Compound 606) to treat trypanosomiasis and

syphilis. His Nobel lecture (1908) was a

detailed description of a theory in which tissue

and chemical side chains were similarly

responsible for the therapeutic effects of drugs

and the protective effects of antibodies. His

theory made the “magic bullet” idea of drug

therapy popular, reinforcing the idea that each

disease has its specific drug remedy.

The power of the drug industry was able to

make that idea central to medicine, and it

shaped thinking about vaccines for the next

century.

Metchnikov’s 1908 Nobel lecture wasn’t

just about phagocytosis, it was an alternative

view of immunity as a pervasive multi-level

process, involving learning and tissue mainte-

nance as well as specific elimination of patho-

gens. It was either ignored or disparaged by

medical researchers until the end of the 20th

century. He said that at the present time, he

knew of only one substance that could assist

the innate immune system, and that was

quinine. Recent studies relating to a single

kind of cell sensor, the Toll-Like Receptors

(TLRs), have found that quinine reduces their

activation of cytokines and antibodies. Angio-

tensin, the covid virus (Sarius and Perlman,

2021), endotoxin, estrogen, and ionizing

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radiation have opposite effects, activating

TLRs and increasing cytokine and antibody

production.

In Metchnikov’s time, chloroquine and

hydroxychloroquine hadn’t been synthesized

yet. Those drugs weren’t introduced because

they were in some way superior to quinine. In

the 1930s, Germany and the US wanted new

drugs to treat malaria, to get around Dutch

control of the world quinine supply, and

produced several synthetic variations of

quinine, including chloroquine. In 1950,

hydroxychloroquine was produced to avoid the

retinal toxicity of chloroquine. Recently,

because of reports that hydroxychloroquine

could be helpful for treating the corona virus

infection, some researchers compared the

effects in vitro of quinine and the two synthet-

ics on human cells (Grosse, et al., 2021). They

found that natural quinine, like the synthetics,

effectively blocked the infection of human

cells by the corona virus, but was safer. The

more toxic chloroquine had previously been

found to have antiviral effect against SARS

coronavirus (Vincent, et al., 2005).

Metchnikov’s developmental, multilevel,

cellular view of immunity was incompatible

with “modern medicine.” The American

Medical Association was founded in 1847 to

promote “scientific” allopathic medicine, and

to suppress the practice of homeopathy,

empirical medicine, herbalism, etc. It opposed

the use of “crude” herbal preparations, and

considered it necessary to find a pure chemical

substance to specifically treat each condition,

such as a microbial infection, a parasite, or,

later, a genetic defect. This theory of medicine

is a perfect match for the pharmaceutical

industry, and for Ehrlich’s analogy of “magic

bullet” chemotherapy to the function of

antibodies.

It was only a little more than 20 years ago

that Jamie Cunliffe’s “morphostasis” and

“damage theory” and Polly Matzinger’s

“danger theory” broke away from the

dominant antibody theory of immunity, which

was based on Macfarlane Burnet’s mechanis-

tic, neodarwinian “clonal deletion” theory.

These new theories were, in effect, extensions

of Metchnikov’s developmental approach in

which immunity is a process of generating and

maintaining the organism. When a cell is

damaged, it releases internal components

including DNA, RNA, and ATP, and these are

recognized by cells that promote repair of the

damage, under the guidance of systemic inter-

actions. Unfamiliar antigens will be eliminated

along with the damaged tissue, by a combina-

tion of many factors, from phagocytes to

antibodies.

A few people had been recognizing the

failure of the clonal deletion theory for a long

time. At an international meeting of

immunologists in 1969 at the University of

Oregon, a young teacher had demonstrated

that the deleted clones could be restored,

giving the original repertoire of antibodies, if

the immune cells were simply cultured at a

higher density than Burnet and Nossal had

used, allowing the cells to interact with each

other, forming a network that was able to

detect, and repair, the deletions.

Although his lecture was attended by only

a few people, and the main speakers acted as

though it hadn’t happened, Niels Jerne appar-

ently noticed it, because he later presented an

alternative to clonal deletion based on his idea

of an “idiotype-antiidiotype network” (Jerne,

1974). By itself, it wasn’t an adequate theory,

but it moved in the right direction. The

damage and danger theories have provided a

way to discard the various Nobel Prize

winning theories of antibodies and their

functions, and to begin seeing immunity and

resistance as holistic processes of the

organism.

The nature of protective reactions has been

obscured by thinking in terms of “the immune

system,” as something distinct from metabolic

and adaptive processes. The various

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“-phylaxes,” starting with skeptophylaxis

(lightning or quick protection), have made it

clear that the endocrine system and metabolic

systems are deeply involved in the adaptive

protections.

Although mast cells and basophils are

usually the main sources of histamine, experi-

ments have shown that under the most extreme

conditions, all of the cells studied produced

histamine. Energy deprivation activates the

formation of histamine, which activates many

“immune” functions, including increasing the

TLRs, and production of cytokines and

antibodies. Histamine should probably be

thought of as one of the signals of danger or

damage. It seems to correspond to the catabolic

phase of an infection.

Although inflammation can occur without

infection, its presence is generally thought to

indicate infection. The corona virus infections

inactivate the ACE2 enzyme, whose functions

include destroying angiotensin II, a basic

promoter of inflammation, so, more than other

kinds of infection, it is a disease of inflamma-

tion. Since inflammation activates the clotting

system, it is a disease of inflammation and

coagulation. Before the WHO declared the

pandemic, China was treating covid infections

with antiinflammatories and anticoagulants.

Outside of China, governments have ordered

doctors not to treat early cases of covid, until

their symptoms were serious enough to require

hospitalization and oxygen treatment. On

October 22, 2020, the FDA approved the use

of remdesivir in covid patients, but remdesivir

is neither antiinflammatory nor anticoagulant;

inflammation is one of its side effects.

Histamine helps to rouse the immune

functions, and promotes inflammation and

coagulation.

In a prolonged excess, it tends to calcify

soft tissues, reducing their functions. Like

serotonin, it activates carbonic anhydrase, and

is activated by estrogen and a reducing

environment. While histamine is a basic

organizing factor in immunity, its effects have

to be blocked for completion of the immune

processes. Increased oxidative metabolism

lowers histamine and inflammation systemi-

cally, for example by lowering the ratio of

estrogen to progesterone.

Although anaphylaxis is usually thought of

as something separate from the immune

response, I think it just represents a quick

entry into the high-histamine state, because of

low energy reserves and slow mobilization of

the hormones of homeostasis. In the 1960s, V.

W. Adamkiewicz showed that reducing blood

glucose with insulin greatly intensified aller-

gic reactions, while increasing blood glucose

prevented deadly anaphylaxis, but weakened

resistance to infection. Energy metabolism

governs the effectiveness of the immune

process, including the avoidance of autoim-

munity (Gaber, et al., 2017).

Metabolic energy is involved in every

aspect of the covid infection, but that has been

denied or ignored because of a traditional,

ideological understanding of immunity. Denial

of obvious facts isn’t a rare thing in the history

of science, but the recent world-wide reactions

to the corona virus epidemics, including crimi-

nalization of the traditional medical practice of

treating symptoms, are unique and completely

outside any scientific, medical, or legal

standards.

Moderna had been working on RNA

vaccines for years when the “novel” corona

virus was identified, and they had been testing

it as a vaccine for corona virus in mice, so

when they received the genetic code by

telephone from China, it took them only a few

hours to produce the vaccine. As they rushed

through the preliminary trials, it supposedly

didn’t occur to anyone that the vaccine might

spread throughout the body, or that it would

infect the lining of blood vessels with the

spike protein, or that the infected endothelial

cells would cause systemic clotting and

inflammation, or that if the spike protein

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reached the lungs it would severely damage

them, or that anti-spike antibodies would be

toxic to the lungs, or that anti-antispike

antibodies would contain an antigen similar to

the spike antigen. They, like Pfizer, were

undoubtedly aware of the risks of the shedding

phenomenon. These facts would imply either

that no one in the vaccine business had studied

physiology or immunology with any attention,

or that their plan—to inoculate everyone in the

world with a highly toxic genetic

material—wasn’t intended to improve public

health.

REFERENCES

Can Med Assoc J. 1963 April 13; 88(15):

806–811. Glycemia and Immune Responses. V.

W. Adamkiewicz.

The intensity of experimental and clinical

immune responses was correlated with the degree

of glycemia of the reacting subject. Hyperglyce-

mias resulting from overdosage with sugars, corti-

sol, adrenaline, or from diabetes inhibit the

anaphylactoid reactions; anaphylaxis, and the

tuberculin reaction; but potentiate infections.

Hypoglycemias resulting from fasting, insulin and

adrenalectomy potentiate the anaphylactoid

reactions, anaphylaxis, and the tuberculin reaction;

but inhibit infections. The hypothesis is proposed

that hyperglycemia inhibits certain antigen-

antibody combinations; this results in an inhibition

of hypersensitivity, but an aggravation of infection.

Proc Natl Acad Sci U S A. 2009 Jul

21;106(29):12145-50. Neuroprotective natural

antibodies to assemblies of amyloidogenic

peptides decrease with normal aging and

advancing Alzheimer's disease. M Britschgi, C E

Olin, H T Johns, Y Takeda-Uchimura, M C

LeMieux, K Rufibach, J Rajadas, H Zhang, B

Tomooka, W H Robinson, C M Clark, A M Fagan,

D R Galasko, D M Holtzman, M Jutel, J A Kaye,

C A Lemere, J Leszek, G Li, E R Peskind, J F

Quinn, J A Yesavage, J A Ghiso, T Wyss-Coray.

Clin Exp Immunol. 2005 Sep;141(3):500-8.

Antibody to severe acute respiratory syndrome

(SARS)-associated coronavirus spike protein

domain 2 cross-reacts with lung epithelial cells

and causes cytotoxicity. Y S Lin, C F Lin, Y T

Fang, Y M Kuo, P C Liao, T M Yeh, K Y Hwa, C

C K Shieh, J H Yen, H J Wang, I J Su, H Y Lei.

Nature Reviews Rheumatology volume 13,

pages 267–279 (2017). Metabolic regulation of

nflammation. Timo Gaber, Cindy Strehl & Frank

Buttgereit.

Ann Immunol (Inst Pasteur) 125 C: 373.

(1974). Towards a network theory of the

immune system. Jerne NK.

Nat Med. 2005 Aug;11(8):875-9. A crucial

role of angiotensin converting enzyme 2

(ACE2) in SARS coronavirus-induced lung

injury. Keiji Kuba, Yumiko Imai, Shuan Rao,

Hong Gao, Feng Guo, Bin Guan, Yi Huan, Peng

Yang, Yanli Zhang, Wei Deng, Linlin Bao, Binlin

Zhang, Guang Liu, Zhong Wang, Mark Chappell,

Yanxin Liu, Dexian Zheng, Andreas Leibbrandt,

Teiji Wada, Arthur S Slutsky, Depei Liu, Chuan

Qin, Chengyu Jiang, Josef M Penninger

Annu Rev Immunol. 1994;12:991-1045.

Tolerance, danger, and the extended family. P

Matzinger.

For many years immunologists have been well

served by the viewpoint that the immune system's

primary goal is to discriminate between self and

non-self. I believe that it is time to change

viewpoints and, in this essay, I discuss the possi-

bility that the immune system does not care about

self and non-self, that its primary driving force is

the need to detect and protect against danger, and

that it does not do the job alone, but receives

positive and negative communications from an

extended network of other bodily tissues.

Cent Eur J Immunol. 2018; 43(4): 466–475.

Natural antibodies–-facts known and unknown.

Joanna Palma, Beata Tokarz-Deptuła, Jakub

Deptuła, and Wiesław Deptuła.

J Theor Biol. 2015 Jun 21; 375: 71–76.

Molecular Mimicry and Clonal Deletion: A

Fresh Look. Noel R. Rose.

Nat Immunol 22, 801–802 (2021). SARS-

CoV-2 takes its Toll. Sariol, A., Perlman, S.

Immunology. 2004 Oct; 113(2): 224–233.

Histamine induces Toll-like receptor 2 and 4

expression in endothelial cells and enhances

sensitivity to Gram-positive and Gram-

negative bacterial cell wall components. Jaya

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Talreja, Mohammad H Kabir, Michael B Filla,

Daniel J Stechschulte, and Kottarappat N Dileepan

Clin Immunol. 2021 Mar; 224: 108665. Do

COVID-19 RNA-based vaccines put at risk of

immune-mediated diseases? In reply to “potential

antigenic cross-reactivity between SARS-CoV-2

and human tissue with a possible link to an

increase in autoimmune diseases.” Rossella Talotta

Virol J. 2005 Aug 22;2:69. Chloroquine is a

potent inhibitor of SARS coronavirus infection

and spread. Martin J Vincent, Eric Bergeron,

Suzanne Benjannet, Bobbie R Erickson, Pierre E

Rollin, Thomas G Ksiazek, Nabil G Seidah, Stuart

T Nichol.

Clin Immunol. 2020 Aug; 217: 108480. Poten-

tial antigenic cross-reactivity between SARS-

CoV-2 and human tissue with a possible link to

an increase in autoimmune diseases. Aristo

Vojdania, and Datis Kharrazian.

Videos, Virus, Vaccines

Steve Kirsch:

https://trialsitenews.com/silicon‐valley‐drug‐developm

ent‐philanthropist‐tours‐vaers‐comparing‐influenzas‐c

ovid‐19‐vaccine‐data/

https://trialsitenews.com/are‐the‐covid‐19‐vaccines‐sa

fe‐and‐effective/

Mike Yeadon:

https://www.brighteon.com/a92e53c2‐8c35‐498a‐ba7

4‐c6ab652dcffa

https://www.bitchute.com/video/9enapGHu7UmI/

Peter McCullough:

https://www.youtube.com/watch?v=QAHi3lX3oGM

https://rumble.com/vk5jd0‐dr.‐peter‐mccullough‐urge

nt‐warning‐about‐poisonous‐jabs‐an‐agonizing‐situa.ht

ml

Dolores Cahill:

https://www.bitchute.com/video/M7tOFVckPlCT/

Robert Malone interview with Jimmy Dore:

https://www.bitchute.com/video/HzNd9t3ipfzV/

Fleming, Novavax, accountability:

Https://www.bitchute.com/video/LzTeejWo9rTr/

Reiner Fuellmich, July 17, 2021:

https://www.bitchute.com/video/XHEvvvveUVJE/

David Knight and Lee Merritt:

https://www.bitchute.com/video/wBXKh4QXEGtu/

Lee Merritt: (D-dimer 60%)

https://www.brighteon.com/eb1b58c0‐d62f‐4229‐90

98‐5fd861118053

Sucharit Bhakdi:

https://factcheckvaccine.com/2021/07/an‐urgent‐me

ssage‐from‐professor‐dr‐sucharit‐bhakdi‐july‐9‐2021/

https://www.brighteon.com/681b0af1‐f34f‐4d3b‐849

a‐40caf675b7d1

Bhakdi Sept 3:

https://www.bitchute.com/video/ry6p1Glnp760/

Mike Adams interviewing Dr. David Martin:

https://www.bitchute.com/video/ZxGqUdGddRPo/

https://www.brighteon.com/309412a4‐65e5‐4bd6‐a7

c9‐52805b5a6b93

Funeral director:

https://rumble.com/vmlz42‐funeral‐director‐john‐olo

oney‐blows‐the‐whistle‐on‐covid.html

Charles Hoffe, a Canadian doctor:

https://www.bitchute.com/video/ijiltpFuhwyk/

Ryan Cole:

https://www.bitchute.com/video/I5IPjw0POAlF/

Ethics Professor:

https://rumble.com/vm8ie1‐ethics‐professor‐gives‐he

artbreaking‐final‐lesson‐on‐refusing‐vaccine‐befor.ht

ml

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